At this time of year, many nonprofit organizations focus on the relief of poverty and hunger. But humans can also suffer T3 hormone poverty and hunger. We can't be truly healthy without "enough" T3 in our bloodstream, our tissues, organs and cells. We can't be healthy with "too much" T3 activity in cells, either. Well-regulated … Continue reading Our wish: May you be blessed with T3 sufficiency
In thyroid disease and therapy, even when TSH is normalized, we can still be genuinely hypothyroid if we do not have enough T3 getting into our thyroid hormone receptors in cells throughout the body. Most people know there's two ways we get T3 into our cells' nuclei: From circulating Free T3, andFrom circulating Free T4 … Continue reading How do we get enough T3 into thyroid hormone receptors?
We all know a "thyroidectomy" is the surgical removal of a thyroid gland. Nobody removes a vital gland without a patient's informed consent. If your medical system and doctor are ethical, you as a patient have the opportunity to weigh the risks and benefits of the procedure and give signed consent. We need to consider … Continue reading Caution: Doctors perform T3-ectomies without our consent
This post follows up on several recent posts on the topic of Reverse T3 (RT3) and our thyroid hormone conversion enzymes, the three deiodinases. Here I'm providing some practical tips about Reverse T3 testing, test interpretation, and where we should be focusing our attention. In these posts, I aim to improve thyroid therapy by debunking … Continue reading Principles and Practical tips for Reverse T3, FT3, FT4
A lot of internet myths and misinformation surrounds Reverse T3 (RT3). Well-meaning people have attributed to the hormone a “T3-blocking” function. However, the enzyme deiodinase type 3 (D3) (NOT Vitamin D3, which is very different) is the main blocker of T3 hormone. Deiodinase type 3 (D3), regulated by the DIO3 gene, is the enzyme that … Continue reading Deiodinase Type 3, not RT3, plays the T3-blocking role